Symptoms can be mild to severe in people infected with the novice coronavirus, and can be diagnosed several weeks ago in weakened immune systems in severely infected patients.
This was revealed in a medical study conducted in the United States.
The Harvard Hoys Medical Institute study found that at least 3.5 percent of critically ill patients had mutations in genes that are part of the antiviral defense.
Similarly, at least 10% of patients were found to develop auto-antibodies, a factor that attacks the immune system instead of fighting the virus.
The study, published in the journal Science, also identified some of the key factors behind the lethality of code 19.
The researchers said that the discovery of harmful antibodies in 101 of the 987 patients in the study was a startling observation, with the results helping to explain for the first time why code is so serious in some people While most people do not care about the virus.
He said that if a person is diagnosed with corona virus, he should be tested for auto-antibodies, which can remove these antibodies from the blood and reduce the severity of the deadly effects.
The research team, in collaboration with experts from around the world, recruited Code 19 patients for its research in February.
At the time, they were looking for young people with severe code 19 severity, to test for immune system weakness in these patients.
For this purpose, the genomes of patients were scanned, especially the sets of 13 genes that affect immunity against influenza.
In healthy people, the molecules of these genes act as the body’s immune system, identifying viruses and bacteria and sounding the alarm so that other immune systems can be activated.
The research team had in the past discovered genetic mutations that interfere with the formation and function of these molecules.
People with these genetic mutations are more vulnerable to certain germs, including influenza.
The research team also found similar changes in patients with Code 19, which they hope will help doctors identify which patients may be at risk for the disease.
It will also help in finding a cure.
In March, the research team began its work to recruit 500 critically ill people from around the world to code 19, and by August that number had risen to more than 1,500 and now to more than 3,000.
Researchers began analyzing patient samples to discover harmful genetic mutations.
The research team found genetic defects in 23 of the 659 patients who had antiviral defenses.
Researchers believe that without these defenses, it would not be possible for patients with code 19 to get rid of the virus, and this led them to a new idea, that other serious patients may have the same problem, but for a different reason.
According to him, some patients’ bodies may be damaged by these molecules, such as antibodies targeting the body in patients with type 1 diabetes.
The experts then analyzed 987 patients with the deadly disease from code 19 and discovered auto-antibodies in at least 101 cases.
These antibodies block the immune system, while code 19 is not seen in patients with mild or mild infections.
Researchers say that this is an invaluable discovery because it will detect these antibodies and predict who might be seriously ill.
The research team also found that these harmful antibodies were present in 94% of men and that they may be more likely to have the lethal severity of code 19 than women.
The team will now work with other treatment specialists and a clinical trial will be launched.
In addition, they will look for genetic factors that may provide protective effects against the disease, and for this purpose, critically ill patients under Code 19 will be made part of the research.
In addition, people who have been exposed to the virus but have not developed the disease will be included.