Health

A common medicine is said to be helpful in making the corona virus as curable as the common cold

A common blood cholesterol-lowering drug can treat the corona virus in the same way as the common cold.

The claim was made in a medical study in the United States.

The study, conducted by Mount Sinai Medical Center in New York, looked at what the virus needs to survive.

Research has shown that the fat that accumulates inside lung cells is the most important component that the virus needs to replicate itself.

Keeping the virus out means that it will be better controlled, and researchers claim that it can reduce the severity of code 19 as much as a common cold.

“We need to understand how the corona virus controls our metabolism, after which we can regain control of the virus and deprive it of the resources it needs to survive,” the researchers said.

Research has shown that using different drugs can help prevent the virus from copying.

They found that the results from the use of fenofibrate, a drug that lowers cholesterol levels, were encouraging, as it helps lung cells dissolve more fat and the environment needed for the corona virus to thrive. Eliminates

During the research in the laboratory, the researchers succeeded in eliminating the virus almost completely during the treatment with this drug in 5 days.

A study published this week by the Cambridge University Press found that at least 3 out of every 4 deaths from corona virus in China were pre-existing.

More than 40 percent of them had high blood pressure and more than 25 percent had heart disease, both of which are linked to high cholesterol.

Now, experts in the US study say that after clinical trials, this cholesterol-controlling drug may be helpful in the fight against code 19.

Vaccines can take a long time to develop, and there is no guarantee that the therapies currently being researched could be helpful in fighting the virus in the near future.

Last month, the British government approved the use of an anti-inflammatory drug, dexamethasone, following a trial.

Earlier this week, research by the Rensselaer Polytechnic Institute in the United States revealed that a blood-thinning drug could potentially help prevent new corona viruses.

The study found that a common drug approved by the US Food and Drug Administration (FDA) could be a powerful tool against code 19.

The virus, which causes code 19, binds to human cells using its surface spike protein and then causes disease.

But research has found that a blood-thinning drug called heparin binds tightly to this spike protein and can help block the onset of the disease.

The study, published in the journal Antiviral Research, said the drug could reduce the risk of contracting code 19, similar to strategies that have been shown to be effective in preventing other viruses, including Zika and dengue. Happening

The researchers said that this method of early intervention can reduce the disease in people who have been diagnosed with the virus but have no symptoms, but we also see it as a major antiviral strategy. have been.

“Of course we want a vaccine, but there are a number of ways to fight the virus, as we have seen in HIV with the right combination of drugs, we can control the disease by then,” he said. Until a vaccine is ready.

In order to infect a cell, a virus first targets a specific target on the surface of the cell, then passes through the cell membrane, enters its genetic instructions, hijacks machinery, and makes copies.

But the virus can easily be tricked into a molecule that takes the form of a cell target for it, if trapped once the virus can be neutralized and it does not infect any cell or free itself. Krapata, while gradually becoming inoperable.

In humans, the new corona virus binds to a receptor called S2, and according to researchers, papyrus is an equally attractive target for the virus.

Researchers have found that the drug binds to the spike protein of the corona virus up to 73 pu moles (a scale of contact between two molecules).

“It’s very tight-knit, it’s millions of times harder than any other antibody, and once the virus is tied up, it can’t get out,” he said.

During the research, the experts looked at the three types of the drug, how tightly they catch the virus, and then used computational models to determine the specific areas where the molecule could bind to the virus.

All the results confirmed that this drug can be helpful in a successful strategy to deceive the virus.

“It’s not the only virus we’re facing during an epidemic, we don’t have the best antivirals, but there is a way forward, we need to understand that,” the researchers said.

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